Detox
4 min read
May 6, 2023
The dark side of vaping

The dark side of vaping

Vaping is often marketed as a safer alternative to smoking or a tool to quit—but growing evidence suggests it carries serious and under-acknowledged health, environmental, and societal risks. From toxic chemical exposure and cardiovascular damage to reproductive harm and environmental waste, e-cigarettes are far from harmless.

The myth of quitting

Vaping is often promoted as a tool to help people stop smoking, yet the data tells a different story. Although 85% of e-cigarette users report starting vaping to quit cigarettes, a 2013 study across four countries published in the American Journal of Preventive Medicine found they were no more likely to quit than those who never vaped. In fact, reports show that vaping often reduces the likelihood of quitting altogether, with around 70% of users becoming dual users—continuing to smoke while vaping.

What’s actually in the vapour

A 2017 Harvard study analysing 24 flavoured e-cigarette brands found that every single one contained at least one aldehyde or flavouring chemical listed as “High Priority” by FEMA or classified by the FDA as Harmful or Potentially Harmful. Despite this, these products are inhaled repeatedly every day.

We also don’t fully understand how common ingredients—propylene glycol, vegetable glycerin, flavourings, and other additives—behave when heated and aerosolised, especially when used 20 to 30 times a day beyond their original FDA approval context.

Lung injury and toxic exposure

The risks became highly visible during the 2019 outbreak of vaping-associated lung injuries, which resulted in 2,807 hospitalisations and 68 deaths in under a year. One major contributing factor was vitamin E acetate—an additive whose harmless-sounding name masked its severe effects when inhaled.

Research from Johns Hopkins examining 56 e-cigarette devices found that many produced aerosols containing potentially unsafe levels of lead, chromium, manganese, and nickel. These metals originate from heated metal coils and are linked to lung, liver, immune, cardiovascular, and neurological damage, as well as certain cancers. The danger is amplified by ethyl maltol, a common artificial sweetener shown to facilitate the transport of heavy metals into cells.

Cardiovascular and clotting risks

Vaping also exposes users to high concentrations of ultra-fine particles and toxins at levels comparable to conventional cigarettes. These particles activate platelets, increasing their tendency to clump together—an important contributor to clot formation and cardiovascular disease.

Research from Poland suggests that chemicals such as acrolein and formaldehyde, created during e-liquid heating, accelerate arterial hardening and narrowing by more than 1.6 times. Studies from the University of California, San Francisco found that daily e-cigarette use doubles the risk of heart attack, while dual users face a fivefold increase compared to non-smokers.

Hormones, fertility, and pregnancy

The effects extend beyond the lungs and heart. A 2022 study found that vaping doubles the risk of erectile dysfunction in men aged 20 and over. A separate 2020 study of 1,221 men showed a 34% reduction in total sperm count among e-cigarette users compared to non-users.

E-cigarettes contain endocrine-disrupting compounds capable of interfering with hormonal balance and reproductive function. In women, a large study involving 71,940 participants found that unfavourable birth outcomes increased by 62% among those who used e-cigarettes during pregnancy.

Immune suppression and hidden contaminants

Vaping weakens the body’s ability to fight infections. A 2018 study published in BMJ Thorax showed that e-cigarette vapour damages alveolar macrophages—key immune cells responsible for clearing pathogens, toxins, and allergens from the lungs.

Additional concerns come from contamination: an analysis of the ten top-selling U.S. brands detected endotoxins above the limit of detection in 23% of samples tested, adding another layer of inflammatory risk.

Beyond health: society and the environment

Beyond physiology, there is a broader cost. The argument that vaping benefits society is weak when considering its environmental impact. Each disposable vape contains a lithium-ion battery; in the UK alone, the lithium discarded annually in vapes could produce around 1,200 electric vehicle batteries.

On a social level, vaping increasingly functions as an emotional crutch—an “adult pacifier” that outsources stress regulation and potentially disrupts brain chemistry and hormonal balance rather than addressing underlying causes.

The real experiment

E-cigarette use continues to grow rapidly, while robust long-term research on its health effects remains limited. What we do know already points to serious risks—many of which mirror or compound those of smoking itself.

Don’t be society’s guinea pig. It’s time to confront the dark side of vaping.

Next news

The paradigm of depression being a disease/disorder has evolved around the concept that neurotransmitters are primarily the root cause.

Yet, dysregulation in this field could very well be a symptom, coping mechanism and signal from a multitude of different issues ranging from inside, as well as outside of the body (as explained in my previous post).

The association of depression solely being linked to low levels of norepinephrine and serotonin is flawed throughout studies. There are many other variables that can result in this outcome.

Several studies indicate that as few as 25% of depressed patients have low levels of neurotransmitters, while paradoxically, some patients have abnormally high levels of neurotransmitters with no history of them ever being low.

Does the placement of depression into the category of disease/disorder attach a greater overwhelming thought process to the word than if we were to label it as a symptom?

One could argue that generally speaking, symptoms are alleviated with greater ease when compared with the disease.

It is easy to allow our identity to be taken hostage by adopting the ownership of depression and succumb to its depths.

Yet when we shift our thinking to understand that depression does not embody us as individuals, nor does it yield an element involved in modeling our identity, we shift our thoughts to a greater sense of self-empowerment.

We can overcome the ‘depression vs self’ mentality by accepting that depression is an adaptive and protective mechanism from the human body, accompanied with respect for the opportunity it brings forth to gain further insight into what needs nurturing.

I understand this is a challenging and delicate shift to take, yet the perception of the experience is paramount to one’s thoughts.

Could we disempower the shackling chains of emotional paralysis, gain buoyancy in the swamp of thoughts and restore vitality into one’s behaviour when depression is approached in the light of a symptom?

Can we utilise it as foresight with direction to what may require attention and care within our body and how we live?

This paradigm shift allows more of a harmonious level of awareness with the experience of depression and what possibilities it may bring.

References:

  1. Asberg M, et al. Arch Gen Psychiatry. 1976
  2. Mol Psychiatry. 2010 March
  3. Della FP, et al. Pharmacol Biochem Behav. 2012
  4. Della FP, et al. Behav Brain Res. 2012
  5. Della FP, et al. Metab Brain Dis. 2013

Mental Health
6 min read
Depression: disease or symptom?
Depression: disease or symptom?
Viewing depression as a symptom rather than an identity can foster self-empowerment, insight, and more personalised paths to healing—shifting focus from labels to understanding what the body and mind may be asking for attention and care.
December 10, 2022

Got plaque?

The conventional approach uses a process known as ‘Triple Therapy’ – this is the combination of two different antibiotics in-conjunction with a proton-pump inhibitor (which lowers HCL).

Not only does this have a low success rate, but it makes the host vulnerable to infection from other pathogens and parasites post-treatment. This is due to decreased stomach acid and a lessened ability to sterilise bacteria within food along with less competitive dominance within the gastrointestinal tract due to the eradication of the microbiota (pathogenic and beneficial species) within the gut, leaving available space to harbour unwanted bacteria.

H. Pylori is starting to develop resistance to antibiotics, making the conventional approach less and less effective – leading to the ‘thought’ process of adding more medication to the mix to overcome this resistance by using ‘Quadruple Therapy’.

The Health Protection Agency reports that the prevalence of H. Pylori antibiotic resistance varies within the UK depending on location, from 20-63% for metronidazole and 4.4-11% for clarithromycin. [11]

This is one factor explaining why as many as 20% of affected individuals have a reoccurring infection even after treatment.

Another factor for reinfection is the presence of H. Pylori in the oral cavity [1–3]; therefore, failure to eliminate H. Pylori from the mouth could lead to gastrointestinal reinfection.

Gingivitis and chronic periodontitis may be associated with H. Pylori infection and considered as a potential reservoir for bacteria. [4,5]

The plaque should be removed professionally, and oral hygiene procedures should be performed along with eradication strategies of H. Pylori [6] to increase the probability of success.

One could argue that an alternative approach yields much less risk to the host when compared with the iatrogenic complications and long-term hindrance from antibiotics (especially given that a single course of antibiotics can disturb the microbiome for unto 2-4 years post-treatment [12]).

The following has been found effective when addressing H. Pylori:

  • Bismuth subcitrate
  • Mastic gum
  • Goldenseal/Berberine
  • DGL Licorice wafers
  • Matula tea

The following foods have also been found beneficial:

  • Coconut oil
  • Manuka honey

All this has to be addressed with the appreciation of humans being a multi-dimensional and bi-directional organism.

Seeking advice from a trusted professional is always recommended when dealing with any health ailments.

References:

  1. Morales-Espinosa R, et al., Oral Microbiol Immunol. 2009
  2. Nguyen AM, et al., Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 1995
  3. Miyabayashi H, et al., Helicobacter. 2000
  4. Gebara EC, et al., J Clin Periodontol. 2006
  5. Dye BA, et al., Am J Public Health. 2002
  6. Eskandari A, et al. Med Oral Patol Oral Cir Bucal.
  7. Abro AH, et al. J Ayub Med Coll Abbottabad. 2011
  8. Saudi J Gastroenterol. 2014
  9. J Gastrointestin Liver Dis. 2011
  10. J Gastroenterol Hepatol. 2011
  11. Surveillance of Helicobacter pylori antibiotic resistance in England and Wales; Public Health England, 2008
  12. Jernberg, C, et al. 2010
Gut Health
6 min read
Pylori, plaque and protocols
Pylori, plaque and protocols
Reducing plaque, addressing oral health, and using targeted non-antibiotic strategies may improve eradication success while lowering long-term risks to the gut and immune system. A whole-body, professional-guided approach is essential.
December 10, 2022

Are you currently exposed to phthalates on a regular basis? Were you exposed during early developmental stages—for example, through maternal exposure such as a mother working in a hair salon while pregnant? Have you noticed symptom improvement after reducing phthalate exposure?

Higher phthalate levels have been associated with a two-fold increase in the rate of endometriosis. Phthalates are present in almost anything fragranced and are widely used in soft plastics, vinyl, cleaning products, nail polish, and perfumes. As early as 2002, environmental groups reported that over 70% of personal care products contained phthalates. Today, according to the Environmental Protection Agency, more than 470 million pounds of phthalates are produced each year.

Phthalates are now officially recognised as reproductive toxins throughout both the European Union and the United States. Animal studies show that rats given high doses of certain phthalates stopped ovulating altogether. Phthalates reduce oestrogen production by ovarian follicles—oestrogen being one of the primary drivers of follicle growth and egg development in both animals and humans. Suppression of oestrogen by follicle cells would be expected to impair follicle growth, helping explain why women with endometriosis often exhibit significantly higher phthalate levels than those without the condition.

Potential sources of exposure are extensive. Plastics can leach into food, particularly when food is packaged while hot or stored in plastic for long periods. Personal care products are a major contributor, including cosmetics, hair products, lotions, infant care products, medications, medical devices, nail polish, and perfumes.

Vinyl products are another source, such as shower curtains, flooring, wallpapers, blinds, diaper mats, rain gear, inflatable mattresses, school supplies, car interiors, and yoga mats. Additional exposures may come from air fresheners, electronics, plastic jewellery, sex toys, and children’s toys.

Given their prevalence and biological impact, understanding and minimising phthalate exposure is an important consideration in hormone and reproductive health.

Women's Health
6 min read
Phthalates and endometriosis
Phthalates and endometriosis
Commonly found in plastics, fragrances, and personal care products, phthalates can interfere with oestrogen production and reproductive function. Reducing exposure may be a meaningful step in addressing hormone-related symptoms.
February 8, 2022
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