Endometriosis

Trauma can lead to long lasting adaptive mechanisms to protect the individual from repeated exposure, usually these evolutionary defensive behavioural patterns continue later in life despite not being coherent to current day stressors. ⁣

This can lead to distorted situational awareness, disconnecting ones responses and actions from perceived ‘normative’ behaviour.⁣

Highly sensitive triggers may be running in the subconscious mind unknowingly to the individual.⁣

Studies show the adaptive response from trauma can extend over 14 generations. This is a huge component contributing to dysfunctional community syndrome and further stigmatisation.⁣

The following are common factors in trans-generational trauma:⁣

• Accidental’ epidemics⁣
• Massacres⁣
• Starvations⁣
• War⁣
• Slavery ⁣
• The removal of people to reserves ⁣

Below are examples of traumatic events:⁣

• African Slavery⁣
• The Holodomor⁣
• The Holocaust⁣
• World War 1⁣
• World War 2⁣
• Dutch Hunger Winter⁣
• Ritualistic Abuse⁣
• Aboriginal Australian Lineage⁣
• Childhood sexual abuse ⁣
• Middle Eastern War⁣

Do you think trans-generational trauma has impacted you in any way?

Are you currently exposed to phthalates on a regular basis? Were you exposed during early developmental stages—for example, through maternal exposure such as a mother working in a hair salon while pregnant? Have you noticed symptom improvement after reducing phthalate exposure?

Higher phthalate levels have been associated with a two-fold increase in the rate of endometriosis. Phthalates are present in almost anything fragranced and are widely used in soft plastics, vinyl, cleaning products, nail polish, and perfumes. As early as 2002, environmental groups reported that over 70% of personal care products contained phthalates. Today, according to the Environmental Protection Agency, more than 470 million pounds of phthalates are produced each year.

Phthalates are now officially recognised as reproductive toxins throughout both the European Union and the United States. Animal studies show that rats given high doses of certain phthalates stopped ovulating altogether. Phthalates reduce oestrogen production by ovarian follicles—oestrogen being one of the primary drivers of follicle growth and egg development in both animals and humans. Suppression of oestrogen by follicle cells would be expected to impair follicle growth, helping explain why women with endometriosis often exhibit significantly higher phthalate levels than those without the condition.

Potential sources of exposure are extensive. Plastics can leach into food, particularly when food is packaged while hot or stored in plastic for long periods. Personal care products are a major contributor, including cosmetics, hair products, lotions, infant care products, medications, medical devices, nail polish, and perfumes.

Vinyl products are another source, such as shower curtains, flooring, wallpapers, blinds, diaper mats, rain gear, inflatable mattresses, school supplies, car interiors, and yoga mats. Additional exposures may come from air fresheners, electronics, plastic jewellery, sex toys, and children’s toys.

Given their prevalence and biological impact, understanding and minimising phthalate exposure is an important consideration in hormone and reproductive health.

The paradigm of depression being a disease/disorder has evolved around the concept that neurotransmitters are primarily the root cause.

Yet, dysregulation in this field could very well be a symptom, coping mechanism and signal from a multitude of different issues ranging from inside, as well as outside of the body (as explained in my previous post).

The association of depression solely being linked to low levels of norepinephrine and serotonin is flawed throughout studies. There are many other variables that can result in this outcome.

Several studies indicate that as few as 25% of depressed patients have low levels of neurotransmitters, while paradoxically, some patients have abnormally high levels of neurotransmitters with no history of them ever being low.

Does the placement of depression into the category of disease/disorder attach a greater overwhelming thought process to the word than if we were to label it as a symptom?

One could argue that generally speaking, symptoms are alleviated with greater ease when compared with the disease.

It is easy to allow our identity to be taken hostage by adopting the ownership of depression and succumb to its depths.

Yet when we shift our thinking to understand that depression does not embody us as individuals, nor does it yield an element involved in modeling our identity, we shift our thoughts to a greater sense of self-empowerment.

We can overcome the ‘depression vs self’ mentality by accepting that depression is an adaptive and protective mechanism from the human body, accompanied with respect for the opportunity it brings forth to gain further insight into what needs nurturing.

I understand this is a challenging and delicate shift to take, yet the perception of the experience is paramount to one’s thoughts.

Could we disempower the shackling chains of emotional paralysis, gain buoyancy in the swamp of thoughts and restore vitality into one’s behaviour when depression is approached in the light of a symptom?

Can we utilise it as foresight with direction to what may require attention and care within our body and how we live?

This paradigm shift allows more of a harmonious level of awareness with the experience of depression and what possibilities it may bring.

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References:

1. Asberg M, et al. Arch Gen Psychiatry. 1976
2. Mol Psychiatry. 2010 March
3. Della FP, et al. Pharmacol Biochem Behav. 2012
4. Della FP, et al. Behav Brain Res. 2012
5. Della FP, et al. Metab Brain Dis. 2013

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