Mental Health
4 min read
December 10, 2022
Mental health: do you care?

Mental health: do you care?

Mental illness represents one of the greatest public health challenges in Australia and globally. Depression, anxiety, and suicide affect millions of people across all ages, with particularly high impact among young adults, men, Indigenous communities, and LGBTQ+ individuals—yet large gaps remain in awareness, detection, and access to care.

Have you suffered challenges to your mental health?

Depression has the third highest burden of all diseases in Australia (13%) [1] and also third globally [2].

Almost half (45%) Australians will experience a mental illness in their lifetime [3].

One in five, about 20%, Australians aged 16-85 experience a mental illness in any year:

  • 11.5% have one disorder and 8.5% have two or more disorders.
  • 14% of Australians suffer from anxiety disorders.
  • 6% depressive disorders [4].

The age with the highest prevalence of mental illness is between 18-24 years of age.

21.2% of adolescents between 15-19 years of age met the criteria for a probable serious mental illness [5].

Six Australians die from suicide every single day, with a further 30 people will attempt to take their own life [6].

Suicide is the leading cause of death for people Australians aged 25-44 and the second leading cause of death for young people aged 15-24 [7].

Australians are more likely to die by suicide than skin cancer.

Indigenous Australians experience double the rate of suicide when compared to non-indigenous Australians [8].

The LGBT community experiences four times the rate when compared with those identified as heterosexual [9].

In 2011, men accounted for 76% of deaths from suicide [10], yet an estimated 72% of males don’t seek help for mental disorders.

54% of sufferers with mental illness do not seek treatment [1], which is only compounded by the delay in treatment due to problems with detection, social stigma and accurate diagnosis.

Individuals with mental illness accessing treatment are half that of people seeking treatment with physical disorders [11].It’s time to raise awareness for mental health.

References

  1. Australian Institute of Health and Welfare. (2014). Australia’s Health 2014. AIHW: Canberra.
  2. World Health Organisation. (2008). The global burden of disease: 2004 update.
  3. Australian Bureau of Statistics. (2009). National Survey of Mental Health and Wellbeing: Summary of Results, 4326.0, 2007. ABS: Canberra.
  4. Kitchener, B.A. and Jorm, A.F. (2009). Youth Mental Health First Aid: A manual for adults assisting youth. ORYGEN Research Centre, Melbourne.
  5. Ivancic, L., Perrens, B., Fildes, J., Perry, Y. and Christensen, H. (2014). Youth Mental Health Report, June 2014. Mission Australia and Black Dog Institute, Sydney.
  6. The Australian Senate. (2010). The Hidden Toll: Suicide in Australia Report of the Senate Community Affairs References Committee. Commonwealth of Australia, Canberra.
  7. http://www.aihw.gov.au/deaths/causes-of- death
  8. Australian Bureau of Statistic. (2012). Australian Health Survey: First Results. ABS, Canberra.
  9. National Mental Health Commission. (2013). A Contributing Life, the 2013 National Report Card on Mental Health and Suicide Prevention. NMHC, Sydney.
  10. Department of Health and Ageing. (2013). National Mental Health Report 2013: tracking progress of mental health reform in Australia 1993 – 2011. Commonwealth of Australia, Canberra.
  11. Commonwealth of Australia. (2010). National Mental Health Report 2010. Canberra, Australia.

Next news

Got plaque?

The conventional approach uses a process known as ‘Triple Therapy’ – this is the combination of two different antibiotics in-conjunction with a proton-pump inhibitor (which lowers HCL).

Not only does this have a low success rate, but it makes the host vulnerable to infection from other pathogens and parasites post-treatment. This is due to decreased stomach acid and a lessened ability to sterilise bacteria within food along with less competitive dominance within the gastrointestinal tract due to the eradication of the microbiota (pathogenic and beneficial species) within the gut, leaving available space to harbour unwanted bacteria.

H. Pylori is starting to develop resistance to antibiotics, making the conventional approach less and less effective – leading to the ‘thought’ process of adding more medication to the mix to overcome this resistance by using ‘Quadruple Therapy’.

The Health Protection Agency reports that the prevalence of H. Pylori antibiotic resistance varies within the UK depending on location, from 20-63% for metronidazole and 4.4-11% for clarithromycin. [11]

This is one factor explaining why as many as 20% of affected individuals have a reoccurring infection even after treatment.

Another factor for reinfection is the presence of H. Pylori in the oral cavity [1–3]; therefore, failure to eliminate H. Pylori from the mouth could lead to gastrointestinal reinfection.

Gingivitis and chronic periodontitis may be associated with H. Pylori infection and considered as a potential reservoir for bacteria. [4,5]

The plaque should be removed professionally, and oral hygiene procedures should be performed along with eradication strategies of H. Pylori [6] to increase the probability of success.

One could argue that an alternative approach yields much less risk to the host when compared with the iatrogenic complications and long-term hindrance from antibiotics (especially given that a single course of antibiotics can disturb the microbiome for unto 2-4 years post-treatment [12]).

The following has been found effective when addressing H. Pylori:

  • Bismuth subcitrate
  • Mastic gum
  • Goldenseal/Berberine
  • DGL Licorice wafers
  • Matula tea

The following foods have also been found beneficial:

  • Coconut oil
  • Manuka honey

All this has to be addressed with the appreciation of humans being a multi-dimensional and bi-directional organism.

Seeking advice from a trusted professional is always recommended when dealing with any health ailments.

References:

  1. Morales-Espinosa R, et al., Oral Microbiol Immunol. 2009
  2. Nguyen AM, et al., Oral Surg Oral Med Oral Pathol Oral Radiol Endod. 1995
  3. Miyabayashi H, et al., Helicobacter. 2000
  4. Gebara EC, et al., J Clin Periodontol. 2006
  5. Dye BA, et al., Am J Public Health. 2002
  6. Eskandari A, et al. Med Oral Patol Oral Cir Bucal.
  7. Abro AH, et al. J Ayub Med Coll Abbottabad. 2011
  8. Saudi J Gastroenterol. 2014
  9. J Gastrointestin Liver Dis. 2011
  10. J Gastroenterol Hepatol. 2011
  11. Surveillance of Helicobacter pylori antibiotic resistance in England and Wales; Public Health England, 2008
  12. Jernberg, C, et al. 2010
Gut Health
6 min read
Pylori, plaque and protocols
Pylori, plaque and protocols
Reducing plaque, addressing oral health, and using targeted non-antibiotic strategies may improve eradication success while lowering long-term risks to the gut and immune system. A whole-body, professional-guided approach is essential.
December 10, 2022

The paradigm of depression being a disease/disorder has evolved around the concept that neurotransmitters are primarily the root cause.

Yet, dysregulation in this field could very well be a symptom, coping mechanism and signal from a multitude of different issues ranging from inside, as well as outside of the body (as explained in my previous post).

The association of depression solely being linked to low levels of norepinephrine and serotonin is flawed throughout studies. There are many other variables that can result in this outcome.

Several studies indicate that as few as 25% of depressed patients have low levels of neurotransmitters, while paradoxically, some patients have abnormally high levels of neurotransmitters with no history of them ever being low.

Does the placement of depression into the category of disease/disorder attach a greater overwhelming thought process to the word than if we were to label it as a symptom?

One could argue that generally speaking, symptoms are alleviated with greater ease when compared with the disease.

It is easy to allow our identity to be taken hostage by adopting the ownership of depression and succumb to its depths.

Yet when we shift our thinking to understand that depression does not embody us as individuals, nor does it yield an element involved in modeling our identity, we shift our thoughts to a greater sense of self-empowerment.

We can overcome the ‘depression vs self’ mentality by accepting that depression is an adaptive and protective mechanism from the human body, accompanied with respect for the opportunity it brings forth to gain further insight into what needs nurturing.

I understand this is a challenging and delicate shift to take, yet the perception of the experience is paramount to one’s thoughts.

Could we disempower the shackling chains of emotional paralysis, gain buoyancy in the swamp of thoughts and restore vitality into one’s behaviour when depression is approached in the light of a symptom?

Can we utilise it as foresight with direction to what may require attention and care within our body and how we live?

This paradigm shift allows more of a harmonious level of awareness with the experience of depression and what possibilities it may bring.

References:

  1. Asberg M, et al. Arch Gen Psychiatry. 1976
  2. Mol Psychiatry. 2010 March
  3. Della FP, et al. Pharmacol Biochem Behav. 2012
  4. Della FP, et al. Behav Brain Res. 2012
  5. Della FP, et al. Metab Brain Dis. 2013

Mental Health
6 min read
Depression: disease or symptom?
Depression: disease or symptom?
Viewing depression as a symptom rather than an identity can foster self-empowerment, insight, and more personalised paths to healing—shifting focus from labels to understanding what the body and mind may be asking for attention and care.
December 10, 2022

Nutrition is the language of our cells –it dictates how we look, feel, function and perform. It is drastically undervalued when compared the uncountable list of benefits proper nutrition can yield. For this reason, I have decided to write about my top choices of foods that support physiological or neurological changes to favour improved training performance. My initial thoughts were to create a ‘top 10 foods’ article but anyone who knows me, will understand that I really love getting into the nitty-gritty detail.

This will be a 5-part series ranging through meats, nuts, seeds all the way through to vegetables and fruits.

Focal Points

The primary focal point for strength performance is to increase neurological drive (supporting dopamine and acetylcholine production) whilst avoiding any suppression (which would be induced from any GABAergic or serotonergic actions). This will optimise drive, motivation, muscle contraction, muscular tone, focus and clarity whilst avoiding any inhabitation or relaxation.

Other key factors to look at when improving performance would be to ensure that there are optimal levels muscle carnosine levels to buffer PH and offset fatigue, support thyroid function, improve blood flow, support DNA formation, strengthening structural connective tissue and activating the correct pathways to supply the body with available energy stores.

For optimum strength performance, you preferably want to avoid carbohydrates pre-training to ensure there is no pump-induced fatigue and to minimise serotonin secretion – therefore fats and proteins are best choices.

This being said, everyone is individual and what will be highly beneficial for one person may not reap similar effects in another due to food intolerances, nutrient status, hormonal levels and genetic code.

Bone broth – do you even bone though?

This is classed as ‘natures multi mineral’. For thousands of years humans have consumed bones, with theories going as far back as Palaeolithic eras. Bone broths contain bio-available minerals (easily absorbed by the body) such as; calcium, magnesium, phosphorus, silicon, sulphur to chondroitin sulphate and glucosamine.

Cooking bones in a broth releases a combination of nutrients that support:

  • Healthy joint mobility and function
  • Collagen biosynthesis
  • Modulating appropriate immune and inflammatory responses
  • Improved gut health
  • Joint health is a key limiting factor on strength and performance. The neurological system will inhibit strength as a self-safety mechanism to protect the joints by preventing any further damage.

One of the most valuable components of bone broth is gelatin, this acts like a soft cushion between bones that helps them move without friction. Gelatin also provides us with building blocks that are needed to form and maintain strong bones; helping take pressure off aging joints and supporting heathy bone mineral density. Weight training can increase bone density, joint health and mobility but only when it is complemented with an appropriate nutrient regime to feed the specific prosperities to do so.

Another key nutrient in bone broth is collagen, this is found in everything from our joints, bones, ligaments and tendons all the way down to the cellular level. Collagen supports the function of chondrocytes, these are small cells found within the cartilage of joints. Chondrocytes produce mucopolysaccharides and the formation of new collagen that keep joints cushioned and lubricated. Research done by the Department of Nutrition and Sports Nutrition for Athletics at Penn State University found that when athletes supplemented with collagen over the course of 24 weeks, there was significant improvements in joint comfort and a decrease in factors that negatively impacted athletic performance. (Clark KL1, Sebastianelli W, Flechsenhar KR, Aukermann DF, Meza F, Millard RL, Deitch JR, Sherbondy PS, Albert A)

Another powerful nutrient that supports joint health and functionality in bone broth is hyaluronic acid; this acts as a “shock absorber” for joints. It holds water and forms a gel-like substance, lubricating and cushioning joints by surrounding tissues found around nerves (predominantly found in the synovial fluid between joints).

Bone broth is also loaded with glycine. This is classed as a ‘conditionally essential’ amino acid. It is used in the synthesis of haemoglobin, creatine, porphyrin, bile salts, glutathione and the nucleotides DNA and RNA. Glycine is involved in gluconeogenesis (the manufacture of glucose), it helps drive glucose into cells acting similar to the function of insulin. ‘When glycine was ingested with glucose, the plasma glucose area response was attenuated by > 50% compared with the response after the ingestion of glucose alone’ (Mary C Gannon, Jennifer A Nuttall, and Frank Q Nuttall). This means that the athlete with be able to have a slighter high carbohydrate intake to restore glycogen levels, support mTOR, mitigate cortisol and improve training performance.

Glycine also assists digestion by enhancing gastric acid secretion. Research found that only certain proteins stimulate gastric acid secretion, glycine being one (Wald A, Adibi SA.) – thus helping protein digestion and assimilation for enhanced recovery from workout to workout.

Bone broths help mediate appropriate immune and inflammatory responses due to the nature of healing the gut wall (due to nutrients such as proline, glutamine, arginine, gelatin and collagen). This will improve the gut walls integrity and prevent intestinal damage and permeability. If the gut wall becomes permeable, undigested food, toxins and pathogens can pass through the intestinal barrier and into the bloodstream, resulting in a hyper-vigilant immune and inflammatory response to address the ‘foreign invaders’. A healthy response is required for appropriate modulation of training-induced inflammation and endocrine function to enable efficient recovery and optimise super-compensation for growth and development.

Last but certainly not least; roughly 66% of neurotransmitter (chemical brain messengers dictating functions throughout the body) production is made within the gut. If there is any distress or down-regulation in the gut, neurotransmitter production will decrease – resulting in a decrease neural drive.

If the gut wall is inflamed, there is high chance that the brains barrier (blood-brain-barrier) will also be inflamed due to inflammatory stimulation from the vagus nerve, a nerve connecting the gut directly to the brain. This will decrease the brains performance and neurotransmitter reception in the brain.

Go hard or go bone.

Learning to love liver

Organs meats are incredibly abundant in B vitamins, these are essential in detoxification, the citric acid cycle (energy production), neurotransmitter conversions amongst many other key functions within the body crucial for training performance and recovery.

Catechol-O-Methyltransferase (COMT) is one of several enzymes that degrade dopamine, epinephrine, and norepinephrine. COMT introduces a methyl group to catecholamines, donated by the nutrient S-adenosyl methionine (SAMe). Nutrients such as 5-methylfolate (bioactive B9) or methylcobalamin (bioactive B12) support the production of SAMe (Goodman JE, et al. Carcinogenesis. 2001). This is crucial for training performance as the accumulation of too much epinephrine or norepinephrine can lead to over-excitation ‘burn out’ from central nervous system (CNS) fatigue.

Vitamin B12 deficiency is associated with elevated levels of homocysteine (a proinflammatory molecule), fatigue, weakness and adrenal dysfunction (consequently leading to poor energy regulation and a lower androgenic profile).

The recommended minimum intake for B12 is 2.4mcg per day, this is easily hit from beef liver which supplies over 70mcg per 100g (now this may seem extremely high when compared to the RDA, due to it’s nature of being a water-soluble vitamin, toxicity levels are far higher, with no signs of oral toxicity from up to 2,000mcg).

Liver also has a considerable amount of folate (roughly 290mcg per 100g). In 100g of beef liver, there is just over 70% of the RDA requirements for folate (sitting around 400mcg) however this is roughly 3 times less than the optimal levels. We must note, folate is different to folic acid – folate is the bioavailable form that the body actually requires rather than the synthetic version, folic acid, which can place your body under greater amounts of stress dependent if you have a defect in the MTHFR gene. Folate is quintessential for proper methylation whilst also assisting with the recycling BH4 back from its used form, BH2 (Coppen A, et al. J Affect Disord. 1989). This is a key co-factor for the conversion of dopamine (amongst other neurotransmitters) and nitric oxide – thus increasing neural drive and performance whilst increasing the delivery of nutrient-rich, oxygenated blood to throughout the body.

Now I am going to talk about nucleotides, this is a classification of nutrients that are very powerful but unfortunately only known by a minority within the strength and performance world. You name it, Liver is incredibly abundant in this nutrient. They form the backbone of the DNA double helix – this is the most basic genetic material found in the nucleus of a cell that helps forms a blueprint of life. Nucleotides serve varied roles in energy metabolism (integral role of fat, protein and carbohydrates metabolism), enzymatic regulations, signal transduction and as structural components of coenzymes.

Dietary nucleotides are needed by certain cells and their functions for cellular regeneration, largely within the gastro-intestinal tract. Epithelial cells are situated within the intestines, their own production is too low to cover there needs for mucosa growth and its repair process (Sanderson IR, et al. J Nutr. 1994). Within the intestinal flora, specific strains are unable to produce their own nucleotides such as bifidobacterium longum (Rossi M, et al. Arch Microbiol. 2000). The health of the gut dictates your recovery capacity, and you can only train as hard as you can recover. This is done through mediating appropriate immune and inflammatory responses whilst optimizing nutrients digestion and assimilation required for new cellular turnover.

Another aspect how nucleotides can drastically support optimal recovery is the formation of lymphocyte and macrophage cells (Coffey RG, et al. Fed Proc. 1985). These cells are required for appropriate immune function and therefore your management of your biochemistry from training induced stress and inflammation. They have a fast proliferation rate which places a very high demand on nucleotide availability, therefore having a bigger pool of nucleotides means that they have a more efficient production rate.

Nucleotides support the oxygenation of blood, as erythrocyte cells are unable to produce their own nucleotides (Micheli V, et al. Quad Sclavo Diagn 1983). This will increase the circulation and flow of blood around the body to optimize performance and nourish muscles and organs with nutrients in order to heal.

Dietary nucleotides have indirect benefits to support other organs and metabolic processes such as liver function, repairing damaged DNA from oxidative stress and support RNA formation required for the formation of new proteins.

Nucleotides have also been shown to reduce post-exercise immunosuppression and hypothalamic-pituitary axis (HPA) axis activation, whilst also lowering cortisol and creatine kinase (J Strength Cond Res. 2016 Feb) therefore favouring a physiologically anabolic state.

Nutrition
6 min read
Top foods for improved training performance
Top foods for improved training performance
Part 1 of a five-part series exploring performance nutrition through a functional lens. This article examines how bone broth and liver support neural drive, joint health, recovery, and metabolic resilience to enhance strength and training performance.
September 10, 2025
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